The role of M cells and the long QT syndrome in cardiac arrhythmias: simulation studies of reentrant excitations using a detailed electrophysiological model

dc.creatorHenry, Hervé
dc.creatorRappel, Wouter-Jan
dc.date2004-07-19
dc.date.accessioned2026-07-07T05:58:34Z
dc.date.available2026-07-07T05:58:34Z
dc.descriptionIn this numerical study, we investigate the role of intrinsic heterogeneities of cardiac tissue due to M cells in the generation and maintenance of reentrant excitations using the detailed Luo-Rudy dynamic model. This model has been extended to include a description of the long QT 3 syndrome, and is studied in both one dimension, corresponding to a cable traversing the ventricular wall, and two dimensions, representing a transmural slice. We focus on two possible mechanisms for the generation of reentrant events. We first investigate if early-after-depolarizations occurring in M cells can initiate reentry. We find that, even for large values of the long QT strength, the electrotonic coupling between neighboring cells prevents early-after-depolarizations from creating a reentry. We then study whether M cell domains, with their slow repolarization, can function as wave blocks for premature stimuli. We find that the inclusion of an M cell domain can result in some cases in reentrant excitations and we determine the lifetime of the reentry as a function of the size and geometry of the domain and of the strength of the long QT syndrome.
dc.identifierhttps://arxiv.org/abs/q-bio/0407027
dc.identifierhttp://arxiv.org/abs/q-bio/0407027
dc.identifierChaos Vol 14 pages 172-182 (2004)
dc.identifierdoi:10.1063/1.1636272
dc.identifier.urihttp://salesiana.dossiersoluciones.com/handle/123456789/88402
dc.subjectTissues and Organs
dc.subjectOther Condensed Matter
dc.subjectPattern Formation and Solitons
dc.subjectOther Quantitative Biology
dc.titleThe role of M cells and the long QT syndrome in cardiac arrhythmias: simulation studies of reentrant excitations using a detailed electrophysiological model
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