Retinoblastoma protein is the likely common effector for distinct anti-aging pathways

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dc.creatorRadulescu, Razvan Tudor
dc.date2007-07-27
dc.date.accessioned2026-07-07T08:20:52Z
dc.date.available2026-07-07T08:20:52Z
dc.descriptionThe multiple worlds of genetically manipulated laboratory organisms such as transgenic mice or worms with certain gene mutations are somewhat reminiscent of parallel worlds in quantum mechanics. So are various models of aging tested in such organisms. In this context, the tumor suppressor p53 has been found to either accelerate or delay aging, the latter, for instance, in conjunction with ARF, another tumor suppressor, as shown very recently. To more easily determine which of these artificial settings comes closest to real life, I discuss here their features in the light of my protein structure-based insights that have led me to propose a physiological anti-aging role for the retinoblastoma tumor suppressor protein (RB) over the past four years.
dc.description4 pages
dc.identifierhttps://arxiv.org/abs/0707.4174
dc.identifierhttp://arxiv.org/abs/0707.4174
dc.identifier.urihttp://salesiana.dossiersoluciones.com/handle/123456789/135194
dc.subjectSubcellular Processes
dc.subjectBiomolecules
dc.titleRetinoblastoma protein is the likely common effector for distinct anti-aging pathways
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