Non-native beta-sheet formation: insights into protein amyloidosis

dc.creatorGuo, Chinlin
dc.creatorLevine, Herbert
dc.creatorKessler, David A.
dc.date2002-03-08
dc.date.accessioned2026-07-07T02:44:39Z
dc.date.available2026-07-07T02:44:39Z
dc.descriptionProtein amyloidosis is a cytopathological process characterized by the formation of highly beta-sheet-rich fibrils. How this process occurs and how to prevent/treat the associated diseases are not completely understood. Here, we carry out a theoretical investigation of sequence-independent beta-sheet formation, based on recent findings regarding the cooperativity of hydrogen-bond network formation. Our results strongly suggest that in vivo beta-sheet aggregation is induced by inter-sheet stacking dynamics. This leads to a prediction for the minimal length of susceptible polymer needed to form such an aggregate. Remarkably, the prediction corresponds quite well with the critical lengths detected in poly-glutamine-related diseases. Our work therefore provides a theoretical framework capable of understanding the underlying mechanism and shedding light on therapy strategies of protein amyloidosis.
dc.identifierhttps://arxiv.org/abs/cond-mat/0203200
dc.identifierhttp://arxiv.org/abs/cond-mat/0203200
dc.identifier.urihttp://salesiana.dossiersoluciones.com/handle/123456789/19015
dc.subjectSoft Condensed Matter
dc.subjectStatistical Mechanics
dc.subjectQuantitative Biology
dc.titleNon-native beta-sheet formation: insights into protein amyloidosis
dc.typetext

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